Is SARS-CoV-2 a virus? If yes, where does it replicate?
Only in the laboratory eukaryotic cell or also in bacterial cells?
Are the bacteria in the microbiome more numerous than our cells? YES!
And does it seem normal to you that a virus passes through the microbiome layer without bacteria interacting with the virus or producing different substances than usual?
And these bacteria controls we performed and demonstrated
🔷 SARS-CoV-2 replicates first in bacteria
🔷 That orofecal transmission is most important precisely because of the bacterial involvement
🔷 That the bacteria produces toxins
🔷 That antibiotics or a combination of antibiotics can stop both replication, transmission, and toxin production and the clinical picture of patients especially in the early stages of the disease.
FDA has no leg to stand on to justify pulling any OTC supplements off of the market. Who needs testing? 😎 Let’s not forget the non-uniformity of Jab batches. What kind of GMP was that?
Quality control of any product is something that we should always interrogate, and it is the one thing that the entire vaccine industry will not discuss openly.
Which is appalling, because there is no way to adequately address concerns about adverse effects or any potential connection with autism without exploring all the ingredients present in the vaccine shot.
Anything that gets put into the body can have an impact on the body. This is undeniable biological fact, yet is consistently dismissed and ignored as a question by the vaccine industry.
I like to keep an eye on independent researchers such as "Anandamide" of the Nepetalactone newsletter who is checking what's in those vials of Pfizer jab, and finding such contaminants as the DNA that was needed to produce the mRNA. (anandamide.substack.com)
“The focus on antibodies has been extremely harmful...”
What I am curious about (and disregarding other immune system defenses), do antibodies developed only from exposure to the Spike protein created from the mRNA shot have any idiotype diversity? And wouldn’t exposure to the wild circulating virus likely create more antibody idiotype diversity whereby mutations would/could be recognized?
I asked ChatGPT something similar and got this answer:
“Yes, it is possible and likely that a person would develop multiple idiotypes against a virus after a viral infection. The immune system is highly diverse and adaptable. When a person is exposed to a virus, their immune system produces a variety of antibodies, each with slightly different idiotypes. This diversity arises from the way antibodies are generated through a process called somatic hypermutation.
Somatic hypermutation allows B cells (a type of immune cell) to produce antibodies with variations in their variable regions. This diversity is beneficial because it increases the chances of producing antibodies that can effectively neutralize the virus. As a result, a person typically develops a repertoire of antibodies with different idiotypes against a single virus. Some of these antibodies may be more effective at binding to and neutralizing the virus than others.
This diversity in idiotypes is a key aspect of the adaptive immune response, ensuring that the immune system can mount a robust defense against a wide range of pathogens, including viruses.”
There is an important aspect of mRNA inoculations that is different from traditional whole virus vaccines: updating the inoculation requires isolating the requisite spike protein, and then rebuilding the mRNA structure around it.
Which effectively makes them new inoculations every time, and should be put through a full battery of clinical trials.
Whatever data is being provided, these "updated" (actually new) shots are certainly not being tested to that degree. Which challenges the propriety of the regulatory system that authorizes their use.
.
To Say That The Covid-19 Shots
Are "The Best Vaccine Ever"
Is Saying A Lot.
None Of Which Is Good.
What Many Herald As "The Greatest Marketing Plan Ever".
May Actually Be The Worst Marketing Idea. Ever.
.
Is SARS-CoV-2 a virus? If yes, where does it replicate?
Only in the laboratory eukaryotic cell or also in bacterial cells?
Are the bacteria in the microbiome more numerous than our cells? YES!
And does it seem normal to you that a virus passes through the microbiome layer without bacteria interacting with the virus or producing different substances than usual?
And these bacteria controls we performed and demonstrated
🔷 SARS-CoV-2 replicates first in bacteria
🔷 That orofecal transmission is most important precisely because of the bacterial involvement
🔷 That the bacteria produces toxins
🔷 That antibiotics or a combination of antibiotics can stop both replication, transmission, and toxin production and the clinical picture of patients especially in the early stages of the disease.
🔷 That the intermediate host is bacteria.
🔷 That mutations are numerous in bacteria
It’s been known since 2020
51:20 Dr Carlo Brogna Presentation
.
DAY 2 - Full Congress and Presentation Slides
Speakers are happy to share their research presentations with you
https://philipmcmillan.substack.com/p/day-2-full-congress-and-presentation
FDA has no leg to stand on to justify pulling any OTC supplements off of the market. Who needs testing? 😎 Let’s not forget the non-uniformity of Jab batches. What kind of GMP was that?
Quality control of any product is something that we should always interrogate, and it is the one thing that the entire vaccine industry will not discuss openly.
Which is appalling, because there is no way to adequately address concerns about adverse effects or any potential connection with autism without exploring all the ingredients present in the vaccine shot.
Anything that gets put into the body can have an impact on the body. This is undeniable biological fact, yet is consistently dismissed and ignored as a question by the vaccine industry.
That's not okay.
True.
I like to keep an eye on independent researchers such as "Anandamide" of the Nepetalactone newsletter who is checking what's in those vials of Pfizer jab, and finding such contaminants as the DNA that was needed to produce the mRNA. (anandamide.substack.com)
CDC COVID data tracker
2023 aug 19 week 844 deaths
2022 aug 20 week 3166 deaths
2021 aug 21 week 12,181 deaths
2020 aug 22 week 6374 deaths
The current COVID numbers are nothing in a nation that had 3,464,231 deaths in 2021, that is 66,620
a week. 42,915 people died in motor vehicle traffic crashes, crickets.
“The focus on antibodies has been extremely harmful...”
What I am curious about (and disregarding other immune system defenses), do antibodies developed only from exposure to the Spike protein created from the mRNA shot have any idiotype diversity? And wouldn’t exposure to the wild circulating virus likely create more antibody idiotype diversity whereby mutations would/could be recognized?
I asked ChatGPT something similar and got this answer:
“Yes, it is possible and likely that a person would develop multiple idiotypes against a virus after a viral infection. The immune system is highly diverse and adaptable. When a person is exposed to a virus, their immune system produces a variety of antibodies, each with slightly different idiotypes. This diversity arises from the way antibodies are generated through a process called somatic hypermutation.
Somatic hypermutation allows B cells (a type of immune cell) to produce antibodies with variations in their variable regions. This diversity is beneficial because it increases the chances of producing antibodies that can effectively neutralize the virus. As a result, a person typically develops a repertoire of antibodies with different idiotypes against a single virus. Some of these antibodies may be more effective at binding to and neutralizing the virus than others.
This diversity in idiotypes is a key aspect of the adaptive immune response, ensuring that the immune system can mount a robust defense against a wide range of pathogens, including viruses.”
There is an important aspect of mRNA inoculations that is different from traditional whole virus vaccines: updating the inoculation requires isolating the requisite spike protein, and then rebuilding the mRNA structure around it.
Which effectively makes them new inoculations every time, and should be put through a full battery of clinical trials.
Whatever data is being provided, these "updated" (actually new) shots are certainly not being tested to that degree. Which challenges the propriety of the regulatory system that authorizes their use.